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176 You Et Al. Predictors Of Transitions From GADA As The Initial Autoantibody To Multiple Autoantib

Predictors of Transitions From GADA as the Initial Autoantibody to Multiple Autoantibodies of Type 1 Diabetes in Children at Risk by a Dynamic Prediction Model - 10022025_679_176_MP280_You_Ped Diab_2025.pdf PdrdYpbg :pshbkd Mpdchbsnpr ne SpZmrhshnmr Dpnl F9C9 Zr sgd HmhshZk 9tsnZmshancw sn Ltkshokd 9tsnZmshanchdr ne Swod 0 ChZadsdr hm Bghkcpdm Zs Phri aw Z CwmZlhb Mpdchbshnm Lncdk Kt Wnt0 DZkZrshm

https://www.teddy.lu.se/sites/teddy.lu.se/files/2025-11/176%20You%20et%20al.%20Predictors%20of%20Transitions%20From%20GADA%20as%20the%20Initial%20Autoantibody%20to%20Multiple%20Autoantibodies%20of%20Type%201%20Diabetes%20in%20Children%20at%20Risk%20by%20a%20Dynamic%20Prediction%20Model%20-%20Ped%20Diab_2025.pdf - 2026-05-15

Functional C1-inhibitor diagnostics in hereditary angioedema: Assay evaluation and recommendations

Hereditary angioedema (HAE) is an autosomal dominant disease characterized by recurrent episodes of potentially life-threatening angioedema. The most widespread underlying genetic deficiency is a heterozygous deficiency of the serine protease inhibitor Cl esterase inhibitor (C1-Inh). In addition to low C4 levels, the most important laboratory parameter for correct diagnosis of HAE or angioedema du